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‘Neurocognitive and social cognitive deficits in patients with anorexia nervosa’
 
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Instytut Psychiatrii i Neurologii
 
 
Submission date: 2013-10-23
 
 
Final revision date: 2014-01-07
 
 
Acceptance date: 2014-02-07
 
 
Publication date: 2014-06-28
 
 
Corresponding author
Dorota Kułakowska   

Instytut Psychiatrii i Neurologii, Sobieskiego 9, 02-957 Warszawa Warszawa, Polska
 
 
Psychiatr Pol 2014;48(3):465-475
 
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Summary In the first part of the article authors present set of the actual concepts explaining problems of cognitive functions and social cognition currently observed in patients with anorexia nervosa (AN). It is possible; through the neuroimaging research, to get better understanding of the brain specifics in these individuals. Even though, the AN remains a disease with very complex and multifactorial etiology which still is a huge medical challenge. Currently, popular is the view, that takes into consideration the integrating role of the insula and subcortical structures (such as hippocampus, amygdala, thalamus) in the regulation of cognitive and emotional processes in people suffering from AN. There is still an open problem, however, of the selection of therapeutic interventions targeting these deficits. The second part of the article presents attempt to describe deficits in neurocognitive and social cognition in people with AN occurring prior to illness, during and after the recovery. Particular attention has been paid to the most frequently described in the literature - neurocognitive deficits such as rigidity of thinking, weak central coherence, and deficits in social cognition, including mental processes of perception and expression of emotions, disorders of the theory of mind (ToM ) and empathy. The results of previous studies, their scarcity in Poland, do not give a satisfactory answer to the question whether the above mentioned disorders are a feature of endophenotype or condition in an episode of the disease. Research points to the more permanent nature, which may be more resistant to therapeutic modifications.
eISSN:2391-5854
ISSN:0033-2674
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